RecruitingPhase 1Phase 2NCT06071013

Nintedanib Plus EGFR TKI In EGFR-mutated Non-small Cell Lung Cancer Patients

A Phase I/Phase II Study of Nintedanib Plus EGFR TKI In EGFR-mutated Non-small Cell Lung Cancer Patients


Sponsor

China Medical University Hospital

Enrollment

20 participants

Start Date

Feb 23, 2024

Study Type

INTERVENTIONAL

Conditions

Summary

The purpose of this study is to evaluate the efficacy and safety of Nintedanib with EGFR-TKI in participants with advanced EGFR-TKI-resistant non-small cell lung cancer


Eligibility

Min Age: 20 YearsMax Age: 70 Years

Inclusion Criteria4

  • Participants between 20 to 70 years old, are pathologically confirmed advanced (stage III and IV) non-small cell lung cancer.
  • Positive EGFR mutations are diagenesis.
  • Participants with histologically/cytologically confirmed locally advanced or metastatic adenocarcinoma subtype NSCLC after the failure of first-line EGFR tyrosine kinase inhibitors- gefitinib, erlotinib, afatinib, or osimertinib.
  • Participants must have adequate hepatic, renal, and bone marrow function

Exclusion Criteria5

  • Participants previously received first-line EGFR tyrosine kinase inhibitor with serious side effects.
  • Participants have known hypertension, and chronic liver and gastrointestinal disease.
  • Participants have known brain metastasis.
  • Female participants who are pregnant or breast-feeding
  • Participants have a known diagnosis of negative nPKCδ expression by immunohistochemistry (IHC).

Interventions

DRUGNintedanib, gefitinib, erlotinib, afatinib, osimertinib

Nintedanib is a multiple tyrosine kinase inhibitor that can bind competitively to the ATP-binding pocket of these receptors and block intracellular signaling, which is critical for the proliferation and migration. Gefitinib, erlotinib and afatinib are the first- and second- generation of EGFR tyrosine kinase inhibitors that reversibly and irreversibly inhibit the binding of ATP to the phosphate-binding loop of ATP binding site in the kinase domain of EGFR, leading to the inhibition of cell proliferation and induction of apoptosis of cancer cells.


Locations(1)

China Medical University Hospital

Taichung, Taiwan

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NCT06071013


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